MOOD DISORDERS
Depression
• depression 2nd in frequency of 1st and 2nd psychiatric
admissions
• what’s first?
• schizophrenia
Depression: Prevalence
• higher if separated or divorced
• women have higher rates
• in about 50% of the cases, the first episode is also
the last
Bipolar
• rapid-cycling has the poorest prognosis
Bipolar vs. Major Depression:
• Bipolar less common
• Bipolar has equal frequency in men and women
• Bipolar is more prevalent among high SES
• Marriage/relationships have no effect on bipolar
– less likely to be depressed if in relationship
• Bipolar has history of hyperactivity in childhood
– depressed have hx of low self-esteem, obsessive thinking,
dependency
• Bipolar depressive episodes more likely to involve psychomotor
retardation, excess sleep
• Course differs
– bipolar episodes are briefer and more frequent
• Bipolar disorder more likely to run in families
Depression: Etiology
• often clear precipitating events for a first episode
of major depressive disorder
• but not for later episodes (diathesis-stress)
Age of onset issues:
• If parent’s age of onset was under 20, lifetime risk
in child twice that if parent’s age of onset were over 30
• If parent’s age of onset occurred after 40, relatives
are at no greater risk than the general population
Behavioral perspective:
• Extinction
• depression as function of:
– inadequate or insufficient reinforcers
– # and range of stimuli that are reinforcing to that person
– availability of such reinforcers in the environment
– person’s skill in obtaining reinforcement
Aversive Social Behavior
• depressives elicit negative reactions from others
• patients with critical spouses more likely to relapse
within 9 months
Behavioral treatment
• Increasing Reinforcement and Social Skills
• Relearning of pleasure - homework
• Teaching social skills
Cognitive perspective:
• Teach “learned optimism”
• “immunization” against depression, in at risk children
• unlearn pessimistic thinking
Seligman study:
• Participants:
• 10-12 years old
• from varied populations
• suffering from mild depression
• or have parents who are in regular conflict
• both risk factors shown to foster later depression
Results
• Follow-up is looking good
• 20% of tx group became more depressed
• as compared to 44% of control group
Hopelessness - Abramson
• lack of control
• condition will persist or recur
• Hopelessness is the best single predictor of suicide
• even better than depression
Negative Schema
• cognitive triad (self, world, future) - Beck
• Reattribution training
• Some studies find cognitive therapy better than drugs
for depression
• most conclude that combination is best
Etiology of Mood Disorders
Biological perspective
Genetics
• 1st degree relatives 1.5-3 times greater risk
• 10 times greater for bipolar
• Suicide runs in families
Twins
• Bipolar MZ 72%, DZ 14%
• Unipolar MZ 40%, DZ 11%
Adoption studies
• bipolar adoptees
• 31% prevalence in biological parents of mood disorders
• as compared to 2% in the biological parents of normal
adoptees
Chromosome 11
• bipolar disorder in Amish families
Hormones
• Dexamethasone Suppression Test
• dexamethasone suppresses cortisol in normal people
• for at least 24 hours
• endogenously depressed excrete high levels of cortisol
• no drug effect while depressed
“non-suppressors”
• tend to not respond to psychotherapy or placebos
• if nonsuppression continues after depressive episode
predicts relapse
Neurotransmitters
Theory:
• increased levels of norepinephrine produce mania
• decreased levels produce depression
• Serotonin decreased in depression
So,
• Decreased serotonin creates predisposition to mood disorders
• given serotonin deficiency, too high NE causes mania,
too low causes depression
Suicide
• CSF of suicide attempters
• particularly violent attempts
• show abnormally low serotonin activity
• link to impulsive, aggressive behavior
Is depression psychobiologically adaptive?
• Depressed people more vigilant
• more accurate in perception of reality?
Medications
MAO inhibitors
• Parnate
• Nardil
• first antidepressants
• MAO enzyme degrades NE and serotonin
adverse side effects
• particularly when combined with “fermented” food (cheese,
wine, beer)
Tricyclics
• Elavil
• Sinequan
• called such due to 3-ringed molecular structure
• block reabsorption of NE and Serotonin
side effects
• drowsiness
• blurred vision
• constipation
• dry mouth
• but milder and no TD
• take 2-4 weeks to take effect
Selective Serotonin Reuptake Inhibitors (SSRIs)
• Prozac
• Paxil
• Zoloft
• block Serotonin reuptake
side effects
• headache
• upset stomach
• anxiety